La considerevole cifra è stata stanziata dalla Parkinson’s Disease Fundation per favorire quattordici progetti di ricerca:
- Epigenetic Dysregulation in Levodopa-Induced Dyskinesia – Thomas Jefferson University, Philadelphia, PA
- Stability of Tetrameric Alpha-synuclein as a Biomarker in Parkinson’s Disease – Harvard Medical School and Brigham and Women’s Hospital, Boston, MA
- Elucidation of the Role of Cholinergic Interneurons in Levodopa-Induced Dyskinesias Using Transgenic Rats and AAV-mediated Overexpression of Modulatable Receptors – Lund University, Lund, Sweden
- Dissecting the Different Properties of Human Alpha-synuclein Between Dopamine and Non-dopamine Neurons in Vivo – University of Chicago, Chicago, IL
- The Cellular Mechanisms of Semaphorin 3E-Plexin-D1 Signaling in Basal Ganglia Circuitry Formation and Neurodegenerative Diseases – Harvard Medical School, Boston, MA
- Identification and Characterization of a Novel Gene for Parkinsonism – Royal Children’s Hospital Parkville, Victoria, Australia
- Interaction of LRRK2 and Tau in Mediating Neurodegeneration in Mouse Models of Parkinson’s Disease – Swiss Federal Institute of Technology in Lausanne (EPFL), Lausanne, Switzerland
- Identifying Connectivity Changes with Deep Brain Stimulation in Parkinson’s Disease – Max Planck Institute for Human Cognitive and Brain Sciences, Leipzig, Sachsen, Germany
- Imaging Impulsive Control Disorders in PD – Toronto Western Hospital, Toronto, Ontario, Canada
- Cyclic GMP Signaling and Experimental Parkinsonism – Rosalind Franklin University, North Chicago, IL
- Optogenetic Characterization of the Pallidostriatal Synapse in Parkinsonian Mice – Northwestern University, Chicago, IL
- Characterization of a Novel Component That Functions Upstream of PINK1 Linking Mitochondrial Unfolded Protein Stress to Mitophagy – Sloan-Kettering Institute, New York, NY
- Role of Parkin in the Clearance of Defective Mitochondria with Deleted mtDNA. A New Mouse Model of Parkinson’s Disease – University of Miami, Miami, FL
- Clearing Defective Mitochondria: How is Parkin Recruited and Activated? – Children’s Hospital Boston, Boston, MA